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Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

TNC+ fibroblasts play a key role in skin inflammation by interacting with T cells.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Activating Notch in adult skin causes T cells and neural crest cells to gather, leading to skin issues.

Alopecia Areata is an autoimmune condition causing hair loss with no cure and treatments that often don't work well.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Alopecia areata involves immune system imbalances that may lead to depression and anxiety.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Tigers had a skin condition causing hair loss and inflammation, but the cause is unknown and treatment didn't work.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Oral lichen planus is a chronic disease causing mouth discomfort and sometimes needs immunosuppressive treatment.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Treg cell-based therapies might help treat hair loss from alopecia areata, but more research is needed to confirm safety and effectiveness.

Older skin has higher cancer risk due to inflammation and stem cell issues.

Early regulatory T cells are crucial for normal skin pigmentation.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Keloid skin disorder involves abnormal fibroblast activation and immune response, linked to a group of genes including FGF11.

Valproic Acid could potentially be used to treat immune-related conditions due to its ability to modify immune cell functions.

Th22 cells are essential for Tβ15-induced hair growth in mice.

The conditions alopecia areata, primary sclerosing cholangitis, and ulcerative colitis may be linked by shared autoimmune and cell death mechanisms.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

Rare changes in the KRT82 gene are linked to a higher risk of Alopecia Areata.