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Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

People with alopecia areata have more Th17 cells and fewer Treg cells, which may be key to the condition's development.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Pure carbon nanotubes are safe for mice, but impure ones cause immune issues and hair loss.

Pregnancy can increase lupus activity, but careful planning and treatment can improve outcomes.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

Graft-versus-host disease is a complication where donor immune cells attack the recipient's body, often affecting the skin, liver, and gastrointestinal tract.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

Hair loss in mice starts with immune cells damaging hair roots before it becomes visible.

Non-immune factors play a significant role in alopecia areata.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.

Low-Level Laser Therapy and other light treatments for hair growth lack strong evidence and need more research.

The conditions alopecia areata, primary sclerosing cholangitis, and ulcerative colitis may be linked by shared autoimmune and cell death mechanisms.

HSP90 and lamin A/C are crucial for hair growth and could be targets for treating hair loss.

Malondialdehyde-modified DNA may trigger an immune response in alopecia areata patients.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Using a 308-nm excimer lamp with minoxidil promotes hair growth in Alopecia Areata patients, especially younger ones or those with smaller bald spots.

Researchers found four genes that could help diagnose severe alopecia areata early.

Screening for iron levels in patients with hair loss may help find a genetic iron overload condition early.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

The document says how to diagnose and treat hair loss from alopecia areata, but there's no cure and treatments vary.

Alopecia areata, a type of hair loss, may be passed through T cells and has genetic links, while treatments vary in effectiveness. Male pattern baldness can be treated with finasteride and is influenced by androgens in hair follicles.

Alopecia areata is an autoimmune disease with genetic links, treatable with certain medications, and can affect mental health.

Alopecia areata may be treated by restoring hair follicle immune privilege and adjusting immune responses.

Alopecia areata can be triggered by specific immune cells without genetic or environmental factors.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

MDSC-Exo can treat autoimmune alopecia areata and promote hair regrowth in mice.

The combination of oral PUVA and corticosteroids helps regrow hair in severe alopecia areata.