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Balding hair follicle cells are smaller, grow less well, and need more effort to culture than non-balding cells.

Cells from balding scalps have more androgen receptors than cells from non-balding scalps.

A substance called DKK-1 increases in balding areas and causes hair cells to die when exposed to DHT.

New genes found linked to balding, may help develop future treatments.

Vitamin C derivative reduces hair loss-related protein in cells.

Found different proteins in balding and non-balding cells, giving insight into hair loss causes.

Androgens may cause hair loss by increasing TGF-beta1 from scalp cells, which inhibits hair cell growth.

Androgens slow hair growth by altering Wnt signaling in balding cells.

TGF-β1 increases androgen receptor activity in hair loss, but Hic-5/ARA55 can counter this effect.

A protein called ectodysplasin-A2 increases a hair growth inhibitor in balding cells, which could be a target for hair loss treatment.

The gene LRRC15 is more active in balding areas of the scalp compared to non-balding areas.

Hair loss in balding individuals is linked to changes in specific hair growth-related genes.

Dexamethasone increases the activity of androgen receptors in human skin cells, which may link it to certain types of hair loss.

Researchers created a cell line to study hair growth and found specific genes affected by dihydrotestosterone.

Scientists created cell lines from balding patients and found that cells from the front of the scalp are more affected by hormones that cause hair loss than those from the back.

High levels of testosterone and 5α-DHT can lead to cell death in cells important for hair growth.

Androgen receptor signaling causes early aging of cells important for hair growth by damaging their DNA.

Oxidative stress affects hair loss in men with androgenetic alopecia.

Certain microRNAs are more common in balding areas and might be involved in male pattern baldness.

Male pattern baldness is linked to higher levels of a certain receptor in the scalp, which leads to the shrinking of blood vessels and hair loss. Early treatment targeting this receptor could be more effective.

Increased cell death and reduced cell growth in hair follicles contribute to baldness.

The research found specific genes that are more active in balding cells, which could be causing hair loss.

Proteins like aPKC and PDGF-AA, substances like adenosine and ATP, and adipose-derived stem cells all play important roles in hair growth and health, and could potentially be used to treat hair loss and skin conditions.

Myotonic Dystrophy may age cells faster, and drugs that target aging could be potential treatments.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Balding scalps show different gene expressions affecting hair growth compared to non-balding scalps.

Oxidative stress contributes to hair graying and loss as we age.

Androgens can both stimulate and cause hair loss, and understanding their effects is key to treating hair disorders.

Male hormones can cause hair loss, but treatments like Minoxidil and Finasteride can help, and targeting TGF-B1 could be a future solution.

Melanoma risk tools need improvement, certain gene mutations cause skin diseases and could be treated by targeting those mutations, skin wrinkling may relate to lung aging due to genetic factors, and oxidative stress affects hair loss but can be reduced in low oxygen.