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The study suggests that a specific gene variation and higher gene activity are linked to increased baldness in Egyptian men.

Hirsute women have lower type 2 17β-HSD enzyme levels, which improve with treatment.

Certain gene patterns in breast cancer are linked to how active hormone receptors are and could affect patient survival.

The androgen receptor gene doesn't affect women with recurrent spontaneous abortions, but having a mutant genotype might protect against it.

Higher androgen levels and site-specific AR expression cause sex-related skeletal differences, and certain steroids can boost AR expression and androgen effects in bone cells.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

The study found that androgen receptors in skin cells mainly affect the focal adhesion pathway and control the caveolin-1 gene, with implications for new treatments for related diseases.

Research from 2011 to 2020 shows androgen receptors could be key for prognosis and treatment in certain breast cancers.

Women with hair loss have more androgen receptors in certain hair follicles.

Minoxidil may work for hair loss by reducing androgen sensitivity and altering hormone-related enzymes.

ERG increases SOX9, promoting prostate cancer growth and invasion.

Multi-omics techniques help understand the molecular causes of androgenetic alopecia.

Androgenetic alopecia is mainly caused by genetic factors and increased androgen activity, leading to hair follicle miniaturization.

The research identified genes and non-coding RNAs in cells that could be affected by testosterone, which may help understand hair loss and prostate cancer.

SA linked to mitochondrial issues and oxidative stress, while AGA involves disrupted hair growth genes.

Hair loss involves immune responses, inflammation, and disrupted signaling pathways.

Reduced AR gene methylation may cause early pubic hair growth in girls.

G allele of AR Stul polymorphism linked to higher hair loss risk, especially in white people.

Certain genetic variations in the AR and ERβ genes can affect androgen levels in women.

A gene called HDAC9 might be a new factor in male-pattern baldness.

Genetic variation in the androgen receptor gene mainly causes early-onset hair loss, with maternal inheritance playing a key role.

Men with hair loss have more DNA changes in back-of-head hair follicles, possibly protecting them from thinning.

The study found that women with hyperandrogenic PCOS have higher levels of AKT1 and AKT2 proteins in their cells, which may lead to cell dysfunction.

Effective treatments for spinal and bulbar muscular atrophy are not yet available; more research is needed.

Using a combination of specific cell cycle regulators is better for safely keeping hair root cells alive indefinitely compared to cancer-related methods.

Mutations in the androgen receptor gene cause Androgen Insensitivity Syndrome, affecting sexual development.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

Mice with human gene experienced hair loss when treated with DHT.

Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.