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SOX9 is essential for the development of various organs and hair follicles.

Cholesterol balance is important for hair health, and problems with it can lead to hair loss conditions.

Eyelid cells share signaling components but differ in pathway activity.

WNT signaling is crucial for skin development and healing.

Wnt activation shows promise for regenerative medicine but requires selective targeting to minimize risks like cancer.

Found different proteins in balding and non-balding cells, giving insight into hair loss causes.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

A substance called miR-1246 may help treat severe hair loss by reducing certain immune cell activities.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

New treatments for skin conditions related to the sebaceous gland are being developed based on current research.

Bead-jet printing of stem cells improves muscle and hair regeneration.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Epidermal Wnt/β-catenin signaling controls fat cell formation and hair growth.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Wnt signaling is crucial for skin and hair development and its disruption can cause skin tumors.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Colorectal cancer development involves both genetic changes and epigenetic alterations like DNA methylation and microRNA changes.

Certain genetic variants linked to immune response increase the risk of alopecia areata in Taiwanese people.

Stress hormone corticosterone blocks a growth factor to slow down hair stem cell activity and hair growth.

The HOXC13 gene affects different hair proteins in cashmere goats in varied ways and is controlled by a feedback loop and other factors.

Distinct β-catenin patterns are linked to cell growth, not cell death, in lung cancer.

Excessive sun protection might cause frontal fibrosing alopecia by disrupting skin immune balance.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

KLF4 is important for maintaining stem cells and has potential in cancer treatment and wound healing.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

Early-stage skin cells help regenerate hair follicles, with proteins SDF1, MMP3, biglycan, and LTBP1 playing key roles.