Search

everythinglearnresearchcommunity

for

Search:↓

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

The HR protein's role as a repressor is essential for controlling hair growth.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Hairless protein can block vitamin D activation in skin cells.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

VDR regulation varies by tissue and is crucial for its biological functions.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

Cholesterol balance is important for hair health, and problems with it can lead to hair loss conditions.

Hormones and neuroendocrine factors control hair growth and color, and more research could lead to new hair treatment options.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Taking vitamin D might improve life for MS patients and reduce skin side effects from alemtuzumab treatment.

Six genetic conditions are often linked to complete scalp hair loss in children.

Low hemoglobin levels are significantly correlated with hair loss in women with Telogen Effluvium.

HVDRR is caused by VDR gene mutations, leading to vitamin D resistance, treatable with high calcium doses, but alopecia remains permanent.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.