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Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Only minoxidil and finasteride are FDA-approved for hair loss, with other treatments available but less effective or with side effects.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

The Hairless gene is crucial for healthy skin and hair growth.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

The vitamin D receptor can work without its usual activating molecule.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

The skin acts like an endocrine organ, producing hormones that affect various body functions and skin health, and understanding this can lead to new treatments.

TGF-β2 plays a key role in human hair growth and development.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

Vitamin D deficiency is common in people with certain types of hair loss, like alopecia areata and female pattern hair loss.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Combining skin tissue pathology with genetics has greatly improved the diagnosis and understanding of certain skin diseases.

Retinoic acid affects skin and hair health by working with specific receptors, and its absence can lead to hair loss and skin changes.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

ΔNp63α helps control a protein that stops cancer cells from spreading.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.

Genetic mutations can cause hair growth disorders by affecting key genes and signaling pathways.

Mutations in certain receptors can cause diseases and offer new treatment options.

Genetic testing confirmed a young girl has Atrichia with Papular Lesions due to mutations in the hairless gene.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

New gene identification techniques have improved the understanding and classification of inherited hair disorders.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Vitamin D receptor helps control hair growth genes in skin cells.