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T cells and bacteria in the gut and skin help maintain health and protect against disease.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Unconventional lymphocytes are important for quick immune responses and healing of skin and mucosal barriers.

The humanized AA mouse model is better for testing new alopecia areata treatments.

Gamma delta T cells in the skin help with healing and defense but can also cause autoimmune issues, and more research is needed to understand how they are activated.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

The document concludes that Borrelia afzelii causes a skin condition in France, a gene is linked to hair loss in Caucasian women, and various genetic mutations affect skin diseases.

Fibroblast Growth Factors (FGFs) are important for tissue repair and regeneration, influencing cell behavior and other factors involved in healing, and are crucial in processes like wound healing, bone repair, and hair growth.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.

T-regulatory cells are important for skin health and can affect hair growth and reduce skin inflammation.

Certain immune cells are linked to non-scarring hair loss, suggesting potential for immune-targeted treatments.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

New treatments for hair loss from alopecia areata may include targeting immune cells, using stem cells, balancing gut bacteria, applying fatty acids, and using JAK inhibitors.

Radiation therapy damages skin structure and immune function, causing inflammation and potential hair loss.

Hair follicle stem cells are promising for wound healing but require more research for safe clinical use.

Using skin stem cells and certain molecules might lead to scar-free skin healing.

Muscles and nerves that cause goosebumps also help control hair growth.

Stem cells help repair tissue mainly by releasing beneficial substances, not by replacing damaged cells.

New therapies for alopecia areata show potential but need more research.

The immune system plays a key role in tissue repair, affecting both healing quality and regenerative ability.

Wnt signaling is important for development and cell regulation but can cause diseases like cancer when not working properly.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

The conclusion is that understanding how hair follicle stem cells live or die is important for maintaining healthy tissue and repairing injuries, and could help treat hair loss, but there are still challenges to overcome.

Hydra can help understand human hair follicle microbiomes and develop new skin disease therapies.

Stem cells and their secretions could potentially treat stress-induced hair loss, but more human trials are needed.

Vitamin A helps skin stem cells decide their function, aiding in hair growth and wound repair.

Autophagy helps keep skin healthy and may improve treatments for skin diseases.