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Understanding how epigenetic regulation affects stem cells is key to cancer insights and new treatments.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

CXXC5 is a protein that controls cell growth and healing processes, and changes in its activity can lead to diseases like cancer and hair loss.

The p53 protein has complex, sometimes contradictory functions, including tumor suppression and promoting cell survival.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

The Wnt/β-catenin pathway is important for body functions and diseases, and targeting it may treat conditions like cancer, but with safety challenges.

Colorectal cancer development involves both genetic changes and epigenetic alterations like DNA methylation and microRNA changes.

Bone Morphogenetic Proteins (BMPs) help control skin health, hair growth, and color, and could potentially be used to treat skin and hair disorders.

Eating less sugar, milk, and saturated fats and more vegetables and fish may help treat and prevent acne.

The research showed how melanocytes develop, move, and respond to UV light, and their stem cells' role in hair color and skin cancer risk.

The JAK/STAT pathway is important in cell processes and disease, and JAK inhibitors are promising for treating related conditions.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

The environment around the time of conception can change the VTRNA2-1 gene in a way that lasts for years and may affect disease risk.

Vitamin D and its receptor may help prevent skin cancer.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Histone demethylases play a key role in the development of many diseases and may be targets for treatment.

Vitamin D receptor may help protect against UV-induced skin cancer.

Runx1 affects hair growth, cancer development, and autoimmune diseases in epithelial tissues.

Finasteride affects SRD5A2 gene pattern, possibly causing lasting side effects.

Gene therapy shows promise for treating skin disorders and cancer, but faces technical challenges.

Cancer can hijack the body's cell repair system to promote tumor growth, and targeting this process may improve cancer treatments.

The congress highlighted new gene therapy techniques and cell transplantation methods for treating diseases.

Nuclear shape and chromatin changes affect gene expression in skin cell differentiation.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

The document reports findings on genetic research, including ethical concerns about genome editing, improved diagnosis of mitochondrial mutations, solving inherited eye diseases, confirming gene roles in epilepsy, linking a gene to aneurysms, and identifying genes associated with age-related macular degeneration.

Understanding where cancer cells come from helps create better prevention and treatment methods.