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Hedgehog pathway inhibitors can treat certain aggressive cancers but face limitations like resistance and side effects.

Tumor suppressors help control inflammation in cancer and restoring their function could lead to new treatments.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

B cells can both help and hinder the body's defense against melanoma.

NF-κB signaling is crucial in many diseases and can be targeted for new treatments.

Combining specific nanoparticles with immune therapy significantly improves cancer treatment.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

Tissue-derived extracellular vesicles are crucial for cancer diagnosis, prognosis, and treatment.

Targeting and modifying the stem cell niche can improve regenerative therapies.

Using protein degradation to fight cancer drug resistance shows promise but needs more precise targeting and fewer side effects.

Markers CRABP1, Nestin, and Ephrin B2 are present in skin cancer environments and may influence their development.

A man with a type of skin lymphoma had unusual yellowish skin growths despite normal blood lipid levels, and treatment reduced some symptoms but not the growths.

The reviewers suggest clarifying and expanding on the role of inflammation in stem cell-related cancer development.

The reviewers suggest major revisions for clarity and inclusion of recent findings on inflammation's role in cancer.

Reviewers suggest clarifying the abstract, adding background on inflammation's effect on cancer, discussing immune cell roles, including counter studies, and correcting grammar.

The revised study on how inflammation affects cancer stem cells was accepted after improving clarity and structure.

The revised manuscript on how inflammation affects cancer stem cells was accepted after addressing issues with clarity and detail.

Mutations in the SHH pathway in certain skin cells can cause skin tumors and abnormal hair growth.

Metformin, a diabetes drug, may help prevent and treat various cancers, but more research is needed.

New pharmaceutical biomaterials, especially nanomaterials, show promise for improving cancer treatment and disease diagnosis.

The conclusion is that accurately replicating the complexity of the extracellular matrix in the lab is crucial for creating realistic human tissue models.

The silk fibroin-based hydrogel shows promise for treating melanoma and healing infected wounds by killing tumor cells and bacteria, and supporting skin recovery.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

The local environment is crucial for cell development in the tongue.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

LED light therapy at 863 nm wavelength can slow down skin tumor growth and reduce inflammation in mice.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.