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The current understanding of frontal fibrosing alopecia involves immune, genetic, hormonal factors, and possibly environmental triggers, but more research is needed for effective treatments.

People with alopecia have a higher risk of thyroid cancer.

T-regulatory cells are important for skin health and can affect hair growth and reduce skin inflammation.

Men are more likely to have severe respiratory viral infections like COVID-19 due to hormonal and genetic differences, while women generally have stronger immune responses.

The document concludes that managing skin conditions during pregnancy is important and requires specialized care.

Quercetin effectively treated and prevented hair loss in mice.

Certain daily habits like stress, diet, and sleep can affect the severity of hair loss in alopecia areata.

Higher CD70 and CD27 gene expression in alopecia areata lesions predicts disease severity and activity.

Grateloupia elliptica extract may help prevent hair loss and promote hair growth.

Mogamulizumab can cause hair loss and skin rashes.

Probiotic bacteria improved skin and hair health in aged mice.

Different types of cell death affect skin health and inflammation, and understanding them could improve treatments for skin diseases.

Adalimumab can cause complete hair loss in rare cases.

High levels of the protein Flightless I worsen ulcerative colitis symptoms in mice.

Dupilumab is being tested for many new skin, respiratory, and gastrointestinal conditions.

Different sPLA2 enzymes affect immunity, skin and hair health, reproduction, and may be potential targets for therapy.

Blocking interferon-gamma might help treat various autoimmune diseases.

Current and future treatments for alopecia areata focus on immunosuppression, immunomodulation, and protecting hair follicles.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

Alopecia Areata might be linked to Crohn's disease.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

Excessive sun protection might cause frontal fibrosing alopecia by disrupting skin immune balance.

Nonantibiotic macrolides show promise for treating various inflammatory skin conditions.

Certain immune-related proteins are higher in people with alopecia and their healthy relatives, hinting at a genetic link.

Stress can worsen skin and hair conditions by affecting the skin's immune response and hormone levels.

Alopecia areata is an autoimmune disease causing patchy hair loss, often with other autoimmune disorders, but its exact causes are unknown.

Stress increases nerve fibers and immune cell activity in mouse skin, possibly worsening skin conditions.