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The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Follicular mycosis fungoides can look like a B-cell lymphoma, making diagnosis difficult.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

The JAK/STAT pathway is important in cell processes and disease, and JAK inhibitors are promising for treating related conditions.

Tofacitinib helped most patients with alopecia areata regrow hair and changes in immune cells were linked to the treatment's effectiveness.

The study suggests that hypothyroidism may cause alopecia areata.

A man developed a skin cancer called folliculotropic mycosis fungoides after a kidney transplant.

Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

Skin lymphoproliferative disorders are hard to diagnose and often linked to systemic diseases, but most have a good prognosis with accurate diagnosis.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Cats likely have a reactive skin condition, while dogs may have a more complex, possibly cancerous one.

T cells and bacteria in the gut and skin help maintain health and protect against disease.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

New treatments targeting T-cell pathways are needed for better alopecia areata management.

Interferon alfa-2a is effective for treating cutaneous T cell lymphoma but has significant side effects.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

Some patients with a type of skin lymphoma can experience a rare, non-scarring hair loss that looks like another hair loss condition but has distinct features.

The growth of the Epstein-Barr virus in the patient's cells was linked to the worsening of her lymphoma.

Vesiculobullous lesions should be considered part of canine cutaneous epitheliotropic T-cell lymphoma.

Treg cell-based therapies might help treat hair loss from alopecia areata, but more research is needed to confirm safety and effectiveness.

A substance called miR-1246 may help treat severe hair loss by reducing certain immune cell activities.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

FoxN1 overexpression in young mice harms immune cell and skin development.

Foxp3 is crucial for regulatory T cell function, and targeting these cells may help treat immune disorders.

The document concludes that computational models are useful for understanding immune responses and could improve cancer immunotherapy.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.