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The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

TSLP affects atopic dermatitis by increasing sebum and reducing fat through IL-4/IL-13 signaling.

Lgr5 is a marker for active, self-renewing stem cells in the intestine and skin, important for tissue maintenance.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

STAT3 signaling is important for healthy skin and hair follicles, and its disruption can lead to skin conditions like atopic dermatitis.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

Ectodysplasin signaling is crucial for skin appendage development, requiring specific doses and durations.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

The study found that a specific signaling pathway helps skin wounds heal faster but may lead to larger scars.

Wnt/β-catenin signaling is important for keeping skin cell attachment structures stable.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

Skin stem cells interacting with their environment is crucial for maintaining and regenerating skin and hair, and understanding this can help develop new treatments for skin and hair disorders.

Vitamin D and its receptor may help prevent skin cancer.

Wnt signaling is important for development and cell regulation but can cause diseases like cancer when not working properly.

The skin's ability to produce hormones is linked to various skin conditions, and better understanding this process could lead to new treatments.

A certain signaling pathway in mice, when increased, causes hair to gray by depleting the cells that give hair its color.

The microenvironment, especially mechanical forces, plays a crucial role in hair growth and could lead to new treatments for hair loss.

Skin cysts might help advance stem cell treatments to repair skin.

Derinat helps extend hair growth by reducing harmful molecules in skin cells.

BMP signaling is important for skin color, affecting melanin production, pigment spread, and cell movement.

Skin stem cells are crucial for maintaining and repairing skin, with potential for treating skin disorders and improving wound healing.

Overexpression of a specific receptor in mice skin causes skin thinning, early skin barrier formation, eye issues, and hair loss.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

The skin protects the body and is constantly renewed by stem cells; disruptions can lead to cancer.

Using skin stem cells and certain molecules might lead to scar-free skin healing.

Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

The article concludes that studying how skin forms is key to understanding skin diseases and improving regenerative medicine.

Eating a high-fat fish oil diet caused mice to lose hair due to a specific immune cell activity in the skin linked to a protein called E-FABP.