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Artificial skin development has challenges, but new materials and understanding cell behavior could improve tissue repair. Also, certain growth factors and hydrogel technology show promise for advanced skin replacement therapies.

Removing the ATR gene in adult mice causes rapid aging and stem cell loss.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Advanced human skin models improve drug development and could replace animal testing.

C/EBPalpha and C/EBPbeta are crucial for normal skin and oil gland cell development in adult mice.

The research created a detailed map of skin cells, showing that certain cells in basal cell carcinoma may come from hair follicles and could help the cancer grow.

The research mapped gene activity in developing mouse skin and found key markers for skin cell types and changes from fetal to early postnatal stages.

Sonic hedgehog signaling is needed for the development of touch-receptor cells in the skin, and the loss of Polycomb repressive complex 2 can lead to more of these cells.

Healthy mitochondria in skin cells are essential for proper hair growth and skin cell interaction in mice.

SOX2 is crucial for skin cell function and hair growth, and it plays a role in skin cancer and wound healing.

ATP-dependent chromatin-remodeling complexes are crucial for gene regulation, cell differentiation, and organ development in mammals.

The document concludes that understanding the sebaceous gland's development and function is key to addressing related skin diseases and aging effects.

Runx1 affects hair growth, cancer development, and autoimmune diseases in epithelial tissues.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Thyroid hormones are important for skin health and changes in them can affect conditions like hair loss and eczema.

TAF4 is important for skin cell growth and helps prevent skin cancer in mice.

Boosting HGF signaling could improve the creation of hair follicles in lab-made skin.

Using fat-derived stem cells with the drug meglumine antimoniate can help control skin disease and reduce parasites in mice with leishmaniasis.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

Scientists have made progress in growing mini-organs and regenerating parts of the skin, with plans to treat hair loss in a future trial.

NGAL indicates abnormal skin cell differentiation.

Nestin-expressing cells turn into a specific type of skin cell in hair follicles during development and in adults.

The Hairless gene is crucial for hair cell development, affecting whether skin cells become hair or skin and oil gland cells.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

Nestin marks cells that can become a specific type of skin cell in hair follicles of both developing and adult mice.

Skin problems can be caused or worsened by physical forces and pressure on the skin.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

Early over-expression of FoxN1 harms immune and skin development.

Skin varies in thickness, color, and features due to complex genetic and cellular processes.