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Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

The research identified genes and pathways important for sheep wool growth and shedding.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Understanding how cells die in the skin is important for treating skin diseases and preventing hair loss.

Researchers developed a new method to deliver molecules to hair follicles to manage hair growth without damaging surrounding skin.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Lichen planopilaris causes permanent hair loss and scarring due to damage to hair follicles and can be mistaken for other hair loss conditions.

The document concludes that the hair cycle is a complex process involving growth, regression, and rest phases, regulated by various molecular signals.

The document concludes that the hair cycle is a complex process involving growth, regression, and rest phases, regulated by various molecular signals.

Certain proteins, caspases-1 and -11, are important in the early development of skin inflammation in mice.

Gata6 is important for protecting hair growth cells from DNA damage and keeping normal hair growth.

Normal human melanocytes can avoid cell death through multiple pathways.

miR-218-5p helps skin and hair growth by targeting SFRP2 and activating a specific signaling pathway.

Exosomes from skin cells can boost hair growth by stimulating a gene called LEF1.

mTORC1 activity is important for hair growth and color, and targeting it could help treat hair loss and greying.

Proper cell death regulation is crucial for normal hair follicle regeneration and skin remodeling.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

The research found changes in gene expression related to cell death in mouse skin that help understand hair follicle development and skin health.

Hair follicles repair 3D injuries using a 2D healing process.

Skin RAGE levels are linked to inflammation and cell death.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

EGFR is essential for normal hair development and follicle differentiation.

Zebrafish are useful for studying and developing treatments for human skin diseases.

Damaged hair follicle stem cells may leave the skin to help maintain youthfulness.

Hair follicle cells help protect against immune attacks by regulating T-cell activity.

Activating δ-opioid receptors can help hair grow.

Inflammation in psoriasis may trigger antimicrobial peptides and cell death.