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Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

FOXC1 boosts SFRP1 in hair loss, suggesting new treatments.

SFRP-4 might be an early indicator of diabetes and hypertension in men with androgenic alopecia.

A protein called sFRP4 from skin cells stops the development of pigment-producing cells in hair.

sFRP4 partially inhibits hair regeneration, but the study needs clearer data analysis and better explanation of the process.

A protein called sFRP4 can partly inhibit hair growth.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

Certain genes controlled by OVOL1 are crucial for creating new hair follicles.

The best long-lasting results in treating hair loss may be achieved through combination therapy, including treatments like finasteride, minoxidil, and platelet-rich plasma injections.

Stress causes hair loss by making the body produce cortisol, which stops hair cells from growing.

Environmental factors at different levels control hair stem cell activity, which could lead to new hair growth and alopecia treatments.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Wnt signaling is crucial for skin and hair development and its disruption can cause skin tumors.

Finasteride and minoxidil work best together for hair loss.

Both immature and mature fat cells are important for hair growth cycles, with immature cells promoting growth and mature cells possibly inhibiting it.

Finasteride exposure affects gene expression and anogenital distance in male rat fetuses.

Hair loss can be caused by stress, infections, drugs, and various diseases, with treatment depending on accurate diagnosis.

Hair follicle stem cells in hairpoor mice are disrupted, causing hair loss.

Lgr5 is a marker for active, long-lasting stem cells in mouse hair follicles.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.

Some wound-healing cells can turn into fat cells around new hair growth in mice.

β-catenin is essential for stem cell activation and proliferation in hair follicles.

The conclusion is that certain cell interactions and signals are crucial for hair growth and regeneration.

Stem cell offspring help control their parent stem cells, affecting tissue health, healing, and cancer.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Androgens can both increase and decrease hair growth in different parts of the body.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.