Search

everythinglearnresearchcommunity

for

Search:↓

The research identified two types of keratinocytes in chicken scales: one for hard scales and another for soft skin, with similarities to human skin differentiation.

Hair growth is controlled by specific gene clusters and proteins, and cysteine affects hair gene expression in sheep.

The KRTAP11-1 gene promoter is crucial for specific expression in sheep wool cortex.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

miR-195-5p reduces hair growth ability in cells by blocking a specific growth signal.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

FOXO1 is important for wound healing, but its dysfunction in diabetes can slow the healing process.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Cornification is the process where living skin cells die to create a protective barrier, and problems with it can cause skin diseases.

Scientists created stem cells that can grow hair and skin.

Disrupting Stat3 in hair follicle stem cells greatly reduces skin tumor formation.

Hair follicle cells resist turning into skin cells.

A mix of tocopherol acetate and L-menthol helps grow hair better than using them separately or using minoxidil.

Calcium reduces involucrin in rat hair bulbs but doesn't affect filaggrin and Kdap.

The research found genes that may protect certain scalp cells from hair loss.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

STAT3 signaling is important for healthy skin and hair follicles, and its disruption can lead to skin conditions like atopic dermatitis.

Apoptosis is essential for human skin development and forming a functional epidermis.

Different types of fibroblasts play various roles in diseases and healing, and more research on them could improve treatments.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

Keratin proteins are crucial for healthy skin, but mutations can cause skin disorders with no effective treatments yet.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

Smart biomaterials that guide tissue repair are key for future medical treatments.

Environmental factors at different levels control hair stem cell activity, which could lead to new hair growth and alopecia treatments.

Chemokine signaling is important for hair development.

Different species and human skin models vary in their skin enzyme activities, with pig skin and some models closely matching human skin, useful for safety assessments and understanding the skin's protective roles.

Genetic research has advanced our understanding of skin diseases, but complex conditions require an integrative approach for deeper insight.

Hair greying is linked to reduced ATM protein in hair cells, which protects against stress and damage.

Faulty inflammasome activation may lead to autoimmune skin diseases and could be a target for new treatments.