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Advancements in tissue engineering show promise for hair follicle regeneration to treat hair loss.

Hair follicle stem cells in hairpoor mice are disrupted, causing hair loss.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.

Dying cells can help with faster healing and new hair growth by releasing a growth-promoting molecule.

A specific enzyme is essential for proper hair follicle stem cell development and healthy skin.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

mTOR signaling helps activate hair stem cells by balancing out the suppression caused by BMP during hair growth.

TPA promotes hair growth by increasing stem cell activity and activating specific cell signals.

New drugs for heart disease may be developed from molecules secreted by stem cells.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Stem cell technologies and regenerative medicine, including platelet-rich plasma, show promise for hair restoration in treating hair loss, but more research is needed.

The conclusion is that teeth, hair, and claws have similar stem cell niches, which are important for growth and repair, and more research is needed on their regulation and potential markers.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

Stress hormone corticosterone blocks a growth factor to slow down hair stem cell activity and hair growth.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Wnt signaling is crucial for pigmented hair regeneration by controlling stem cell activation and differentiation.

Stem cell niches are crucial for regulating stem cell behavior and tissue health, and their decline can impact aging and cancer.

Environmental cues can change the fate and function of epithelial cells, with potential for cell therapy.

A protein called desmoglein 3 is important for keeping hair follicle stem cells inactive and helps in their regeneration.

The hair follicle bulge is an important area for adult stem cells involved in hair growth and repair, with potential for medical use needing more research.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

Mouse hair follicle stem cells were successfully isolated and used to regenerate hair follicles with two different methods.

Different types of skin cells play specific roles in development, healing, and cancer.

Hair follicle stem cells are key for hair and skin regeneration, can be reprogrammed, and have potential therapeutic uses, but also carry a risk of cancer.

Most hair follicle stem cells do not protect their DNA by dividing it unevenly.

The Bcl-2 protein is important for keeping hair follicle stem cells working and preventing hair loss.

LGR5 helps maintain corneal cell characteristics and prevents unwanted changes by controlling specific cell signaling pathways.

sPLA2-IIA increases growth in hair follicle stem cells and cancer cells, suggesting it could be targeted for hair growth and cancer treatment.

HSPGs help control stem cell behavior, affecting hair growth and offering a target for hair loss treatments.