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The right cells and signals can potentially lead to scarless wound healing, with a mix of natural and external wound healing controllers possibly being the best way to achieve this.

Testosterone may slow down wound healing and increase inflammation.

Injecting alpha-melanocyte-stimulating hormone in mice improved skin healing and reduced scarring.

Hair follicle-derived extracellular vesicles may help heal chronic wounds as effectively as those from adipose tissue.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

Nitric oxide gel helps heal skin burns faster by improving skin growth, hair regrowth, and blood vessel formation.

Mice lacking fibromodulin have disrupted healing patterns, leading to abnormal skin repair and scarring.

Mesenchymal stem cell proteins in a special gel improved healing of severe burns.

Self-assembling RADA16-I hydrogels with bioactive peptides significantly improve wound healing.

TGF-β signaling is essential for new hair growth after wounds.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Aloe vera fermentation helps heal burns faster by reducing inflammation and changing gut bacteria.

Magnesium oxide-infused membranes help heal wounds faster by reducing inflammation and promoting skin and hair follicle growth.

Probiotic-coated silk/alginate scaffolds help heal wounds faster and with less scarring.

Wnt1/βcatenin signaling is crucial for heart repair after injury.

Fat stem cells from diabetic mice can help heal skin wounds in other diabetic mice.

Turning scar-forming cells into fat cells can reduce scarring.

Reductive stress messes up collagen balance and alters cell signaling in human skin cells, which could help treat certain skin diseases.

Enterococcus faecalis delays wound healing by disrupting cell functions and creating an anti-inflammatory environment.

Treprostinil, a drug, can delay wound healing in healthy cells but doesn't affect diabetic foot ulcer cells, suggesting further research could help understand its role in treating these ulcers.

4-aminopyridine helps skin wounds heal faster and better.

γδ T cells help with hair growth during wound healing in mice.

ADSCs help in wound healing and skin regeneration but need more research for full understanding.

Skin fat cells help with skin balance, hair growth, and healing wounds.

Chitosan nanofiber scaffolds improve skin healing and are promising for wound treatment.

Twist2 is essential for proper skin healing and hair growth in developing mice.

Foxn1 is important for fat development, metabolism, and wound healing in skin.

Human skin xenografting could improve our understanding of skin development, renewal, and healing.

FGF9 from certain T cells helps create new hair follicles during wound healing, which could potentially be used for hair loss treatments.

A substance called FGF9 from certain immune cells can trigger new hair growth during wound healing in mice, but humans may not have the same response due to fewer of these cells.