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A new mutation in the XEDAR gene might cause a rare skin condition called hypohidrotic ectodermal dysplasia.

Researchers found WNT10A to be a key gene in developing goat hair follicles.

Finasteride exposure affects gene expression and anogenital distance in male rat fetuses.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Menopause causes significant changes in the vaginal introitus, but less so in the labia majora, which may lead to symptoms of vaginal atrophy.

The length of CAG repeats in the androgen receptor gene is linked to ovarian reserve but does not affect how the ovaries respond to stimulation.

Mongolian gerbils heal wounds differently than mice, with unique protein levels and gene expression that affect skin repair.

The research linked PLCD1 gene variants to the development of trichilemmal cysts.

ELL is crucial for gene transcription related to skin cell growth.

Keratin protein production in cells is controlled by a complex system that changes with cell type, health, and conditions like injury or cancer.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.

Camellia japonica extract may improve scalp health and promote hair growth.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

The congress highlighted new gene therapy techniques and cell transplantation methods for treating diseases.

The Msx2 gene affects feather development in Hungarian white geese and a specific gene variation could indicate feather quality.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Genes related to calcium signaling and lipid metabolism are important for curly hair in Mangalitza pigs.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

Deleting the CRIF1 gene in mice disrupts skin and hair formation, certain proteins affect hair growth, a new compound may improve skin and hair health, blood cell-derived stem cells can create skin-like structures, and hair follicle stem cells come from embryonic cells needing specific signals for development.

The document concludes that understanding hair biology is key to treating hair disorders, with gene therapy showing potential as a future treatment.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Mutations in the LSS gene cause a rare type of hereditary hair loss.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

Rabbits lacking the Hoxc13 gene show similar hair and skin issues to humans with ECTD-9, making them good for research on this condition.

A gene called Gk5 controls lipid production in the skin and affects hair growth.