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TET enzymes are important for skin and hair development by controlling gene activity in specific areas.

Genetic variants linked to ten skin diseases were found, showing both immune and non-immune factors play a role.

Certain immune system proteins are important for skin healing but can cause problems if there are too many of them.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Epidermal stem cells are diverse and vary in activity, playing key roles in skin maintenance and repair.

The protein DDX6 helps keep skin cells renewing properly by controlling the production of certain other proteins and breaking down those that cause cells to mature too quickly.

Dermal-epidermal interactions are crucial for hair growth and maintenance.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

MOF controls key genes for skin development by regulating mitochondrial and ciliary functions.

MOF controls skin development by regulating genes for mitochondria and cilia.

Different types of stem cells and their environments are key to skin repair and maintenance.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

The exact identity of skin stem cells and how skin cells differentiate is not fully known.

Telomere damage affects skin and hair follicle stem cells by messing up important growth signals.

ASH2L is essential for skin and hair development.

EZH2 is essential for hair growth and skin cell development.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Mitochondrial reactive oxygen species are essential for skin and hair development.

Vitamin D Receptor is crucial for normal skin and hair growth.

Notch signaling is essential for healthy skin and hair follicle maintenance.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

Skin stem cells are maintained by signals from nearby cells and vary in their ability to renew and mature.

Vitamin D and calcium are important for quick and effective skin wound healing.

Skin may help in getting rid of excess iron through the process of skin cell renewal.

Overexpression of activin A in mice skin causes skin thickening, fibrosis, and improved wound healing.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.