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Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

A protein called MPZL3 in mitochondria slows down hair growth and could be a target for treating hair growth disorders.

Thyroid disease can cause skin, hair, and nail problems, and treating the thyroid condition often improves these symptoms.

Age-related hair loss is linked to the decline and dysfunction of hair follicle stem cells.

Lrig1 could be a marker for advanced sebaceous carcinoma.

PPAR agonists show promise for skin conditions but need more research before being a main treatment.

Murine hair has specific types of ceramides and glucosylceramides but lacks acylceramides.

FA2H is essential for normal fur and sebum production in mice.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

Understanding keratinization is crucial for treating skin conditions like ichthyoses and psoriasis.

Understanding how androgens and their receptors work can lead to improved treatments for skin diseases.

GMG-43AC may help reduce unwanted hair growth and treat certain hair loss conditions.

UV radiation may cause DNA changes in skin, certain UVB therapy helps psoriasis, a new gene mutation is linked to mild piebaldism, different immune cells affect psoriasis, a drug promotes hair growth, and some cancer drugs could treat skin barrier issues.

Sex steroids, especially progesterone, can slow down the growth of mouse melanoma cells.

T-regulatory cells are important for skin health and can affect hair growth and reduce skin inflammation.

Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Different types of sun exposure damage skin cells and immune cells, with chronic exposure leading to more severe and lasting damage.

Scientists created keratinocyte cell lines from human hair that can differentiate similarly to normal skin cells, offering a new way to study skin biology and diseases.

An American Bully with a genetic skin condition improved significantly with specific topical treatments.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Hair keratins evolved from ancient proteins, diversifying through gene changes, crucial for forming claws and later hair in mammals.

Knowing how skin works and its diseases helps doctors diagnose and treat skin conditions better.

The skin and thymus develop similarly to protect and support immunity.

The fascial layer is a promising new target for wound healing treatments using biomaterials.

The document concludes that a better understanding of cell changes during wound healing could improve treatments for chronic wounds and other conditions.

Fat cells help recruit healing cells and build skin structure during wound healing.

Skin bacteria, specifically Streptococcus and Staphylococcus, help in hair regrowth after skin injury and speed up wound healing.

Current therapies cannot fully regenerate adult skin without scars; more research is needed for scar-free healing.

Skin aging is a complex process influenced by various factors, leading to wrinkles and sagging, and should be considered a disease due to its health impacts.