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Potential therapeutic targets for scarring hair loss are identified.

SFRP2 and PTGDS may be key factors in female hair loss.

The research suggests that SFRP2 and PTGDS proteins might be indicators of female pattern hair loss and could contribute to hair loss.

ILC1-like cells may contribute to hair loss in alopecia areata.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Patients with Systemic Sclerosis have much higher levels of GDF-15, which could help predict organ involvement and guide treatment.

A high-fat diet worsens neutrophilic folliculitis by increasing certain chemokines.

Blocking certain pathways with kinase inhibitors may help treat cutaneous lupus erythematosus.

Mogamulizumab can cause hair loss and skin rashes.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

Targeting CD200 could be a new treatment for rheumatoid arthritis.

Treg dysfunction is linked to various autoimmune skin diseases, and understanding Treg properties is key for new treatments.

CBD can either promote or inhibit hair growth depending on the concentration used.

Blocking CCR5 can prevent and improve hair loss in alopecia areata.

Scarred skin in lichen planopilaris loses immune cells due to a decrease in a specific protein in skin cells.

Human hair follicles have a unique way of using energy and might use the Cori cycle; blocking CCR5 could help treat hair loss.

Estrogen increases a growth factor in hair cells which might affect hair loss.

The document concludes that better targeted treatments are needed for wound healing, and single-cell technologies may improve cell-based therapies.

Lichen Planus is a less common condition affecting skin and mucous membranes, with various types and associated risk factors, challenging to diagnose, significantly impacts life quality, and may have a risk of cancerous changes in oral lesions.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

The document concludes that while there are various treatments for Alopecia Areata, there is no cure, and individualized treatment plans are essential due to varying effectiveness.

New understanding of the causes of primary cicatricial alopecia has led to better diagnosis and potential new treatments.

Special immune cells called Regulatory T cells help control skin inflammation and repair in various skin diseases.

Blocking JAK/STAT pathways can help treat hair loss from alopecia areata.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.