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The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Hair follicle cells change their DNA packaging during growth cycles and when grown in the lab.

BRG1 is essential for skin cells to move and heal wounds properly.

Jumonji genes are important for development and their mutations can cause abnormalities, especially in the heart and brain.

Hair follicle hyperplasia is common in both benign and malignant skin lymphoproliferative disorders, with a proposed new term "pseudolymphomatous adnexitis."

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

Different signals work together to change gene activity and guide hair follicle stem cells to become specific cell types.

A second domain of high sulfur KAP genes on chromosome 21q23 is crucial for hair structure.

Nuclear shape and chromatin changes affect gene expression in skin cell differentiation.

The research concluded that hyaluronic acid affects the formation and growth of hair follicle-like structures in a lab setting.

Testosterone applied to rabbit skin increases skin cell growth and changes skin structure.

Newly divided skin cells quickly move to join skin structures due to tissue tension and specific signals.

The experiment showed that human skin grown in the lab started to form early hair structures when special cell clusters were added.

The research reveals how early embryonic mouse skin develops from simple to complex structures, identifying various cell types and their roles in this process.

Aging causes sweat glands to shrink, leading to skin issues, and blue light can help hair grow.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

The p53 protein has complex, sometimes contradictory functions, including tumor suppression and promoting cell survival.

Stem cell differentiation is crucial for skin barrier maintenance and its disruption can lead to skin diseases.

Telogen is an active phase with important biological processes, not a resting phase.

The research suggests that autophagy-related genes might play a role in causing alopecia areata.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.

Porphyra-334 may help reduce wrinkles and promote hair growth.

Key genes can rewire networks, changing skin appendage types.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

Hairless mammals have genetic changes in both their protein-coding and regulatory sequences related to hair.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

Hairless protein affects hair follicle structure by regulating the Dlx3 gene.

Radiation therapy damages skin structure and immune function, causing inflammation and potential hair loss.