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Research 31–60 of 1000+
- PD59-09 PHOSPHODIESTERASE INHIBITORS COULD REVERSE THE PROLIFERATION OF BPH EPITHELIAL CELLS INDUCED BY CD8+ T CELLS IN LOW ANDROGEN CONDITION
- PD23-01 INHIBITOR OF 5A-REDUCTASE TYPES II PROMOTES THE INFILTRATION OF CD8+T CELLS IN BPH TISSUES.
- Soluble CD83 improves and accelerates wound healing by the induction of pro-resolving macrophages
- Folliculotropic CD8+ mycosis fungoides associated with diffuse mucosal involvement
- Pathogenic CD8+ T cells target K71+ Henle’s layer by forming cytolytic immune synapses in Alopecia Areata
- Soluble CD83 reverses alopecia areata via IDO-mediated Treg cell activation
- 060 Heterogeneity and lineage development of memory CD8+ T cells after viral infection of skin
- 068 Pathogenic Th17 cells, CD8+CD69+CD49a- tissue-resident memory T cells and common γ chain receptor + natural killer cells express more IL-17, compared to IFN-γ, under the foxp3+ memory regulatory T cells-depleted microenvironment in patients with chronic alopecia areata
- Alopecia areata is driven by cytotoxic T lymphocytes and is reversed by JAK inhibition
- Alopecia Areata
- Alopecia areata
- Alopecia Areata in Aging C3H/HeJ Mice
- The autoimmune basis of alopecia areata: A comprehensive review
- Skin CD4+ memory T cells exhibit combined cluster-mediated retention and equilibration with the circulation
- Autoimmunity: Alopecia Areata
- Hair follicle immune privilege and its collapse in alopecia areata
- Alopecia areata: Animal models illuminate autoimmune pathogenesis and novel immunotherapeutic strategies
- The Role of Hair Follicle Immune Privilege Collapse in Alopecia Areata: Status and Perspectives
- Understanding autoimmunity of vitiligo and alopecia areata
- Controlled Delivery of T-box21 Small Interfering RNA Ameliorates Autoimmune Alopecia (Alopecia Areata) in a C3H/HeJ Mouse Model
- CXCR3 Blockade Inhibits T Cell Migration into the Skin and Prevents Development of Alopecia Areata
- Vitiligo: An Autoimmune Skin Disease and its Immunomodulatory Therapeutic Intervention
- The role of lymphocytes in the development and treatment of alopecia areata
- Alopecia areata is characterized by dysregulation in systemic type 17 and type 2 cytokines, which may contribute to disease‐associated psychological morbidity
- Frontiers in alopecia areata pathobiology research
- The PDE4 inhibitor, apremilast, suppresses experimentally induced alopecia areata in human skin in vivo
- Inhibiting Janus kinases to treat alopecia areata
- Pathomechanisms of immune-mediated alopecia
- Selective inhibition of JAK3 signaling is sufficient to reverse alopecia areata
- Alopecia Areata in C3H/HeJ Mice Involves Leukocyte-mediated Root Sheath Disruption in Advance of Overt Hair Loss