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Mutations in the SRD5A3 gene cause a new type of glycosylation disorder by blocking the production of a molecule necessary for protein glycosylation.

Free fatty acids in sebum boost skin's defense against acne by increasing antimicrobial peptides.

Alopecia areata in these mice is inherited, more common in young females, and can be treated with triamcinolone acetonide.

Alopecia areata is caused by the immune system attacking hair follicles.

Telocytes might help with skin repair and regeneration.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

The conclusion is that Treg-targeted therapies have potential, but more knowledge of Treg biology is needed for effective treatments, including for cancer.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

We need more research to better understand human hair follicle stem cells for improved treatments for hair loss and skin cancer.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

Scientists created stem cells that can grow hair and skin.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

Human hair follicle stem cells can be isolated using specific markers for potential therapeutic use.

The document concludes that understanding the genes and pathways involved in hair growth is crucial for developing treatments for hair diseases.

Using human fat tissue derived stem cells in micrografts can safely and effectively increase hair density in people with hair loss.

The article concludes that while we understand a lot about how melanocytes age and how this can prevent cancer, there are still unanswered questions about certain pathways and genes involved.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Alopecia areata may be treated by restoring hair follicle immune privilege and adjusting immune responses.

Researchers created a fully functional, bioengineered skin system with hair from stem cells that successfully integrated when transplanted into mice.

Alopecia areata can be triggered by specific immune cells without genetic or environmental factors.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Women with PCOS have fewer activated T cells in their ovarian follicles, which might affect fertility.

Sox2-positive dermal papilla cells have unique characteristics and contribute more to skin and hair follicle formation than Sox2-negative cells.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Epidermal stem cells could lead to new treatments for skin and hair disorders.

Scientists found a gene in mice that causes early hearing loss.