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Vitamin D3 and its analogues affect skin cell growth and development depending on cell density, calcium levels, and serum presence.

People with Alopecia areata often have lower Vitamin D levels, which might affect the severity of their condition.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

A vitamin D receptor mutation causes rickets and affects immune responses.

Certain variations in the vitamin D receptor gene are linked to an increased risk of polycystic ovary syndrome.

Vitamin B6 reduced hair loss in women with diffuse alopecia, but calcium pantothenate had no clear positive effect.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

Vitamin D levels in hair don't match those in blood, so hair isn't a reliable indicator of Vitamin D status.

Vitamin D and its receptor are important for hair and scalp health and may help treat hair loss.

Kids with alopecia areata had lower vitamin D levels than healthy kids.

Students from the Middle East and Moscow have different hair element levels due to their environments.

High prolactin and low vitamin D levels may be linked to hair loss in women.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Low vitamin D levels may affect ovulation and progesterone in infertile women.

Many people in Saudi Arabia use vitamin D supplements, but few experience toxicity, showing the need for professional guidance when taking these supplements.

Children with alopecia areata often have low vitamin D, especially if they have darker skin, it's not summer, or they're not White.

Low iron levels are linked to premature graying of hair, but vitamin D levels are not.

Vitamin D is essential for bone health and may reduce risks of several diseases.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

Balanced vitamin D levels are crucial to delay aging and prevent related diseases.

Hairless protein can block vitamin D activation in skin cells.

European guidelines recommend regular eye and ear exams, skin care, vitamin D supplements, and cautious use of medications for managing congenital ichthyoses.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

The vitamin D receptor can work without its usual activating molecule.

Hair thinning causes stem cell loss through a process involving Piezo1, calcium, and TNF-α.

Vitamin D levels do not affect the risk of developing alopecia areata.

Adding a specific gene to skin cells can help treat skin disorders like psoriasis.