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Collagen makes skin stiff, and preservation methods greatly increase tissue stiffness.

The research reveals how early embryonic mouse skin develops from simple to complex structures, identifying various cell types and their roles in this process.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

Vitamin A helps skin stem cells decide their function, aiding in hair growth and wound repair.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

Embryonic and adult stem cells are valuable for improving skin grafts and cell therapy.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

C/EBPalpha and C/EBPbeta are crucial for normal skin and oil gland cell development in adult mice.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

Mice lacking fibromodulin have disrupted healing patterns, leading to abnormal skin repair and scarring.

Three specific proteins can turn adult skin cells into hair-growing cells, suggesting a new hair loss treatment.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

Epidermal stem cells have potential for personalized regenerative medicine but need careful handling to avoid cancer.

The document concludes that mouse models are helpful but have limitations for skin wound healing research, and suggests using larger animals and genetically modified mice for better human application.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Scientists have found a way to create hair follicles from skin cells of newborn mice, which can grow and cycle naturally when injected into adult mouse skin.

Small molecule DMF improves psoriasis and multiple sclerosis, adult skin cells can be made to grow new hair, certain skin cells initiate hair growth, IL-17C controls gut health and can cause skin inflammation, and skin cells produce IL-17 that can lead to psoriasis.

Stem cells rearrangement regenerates functional hair follicles, potentially treating hair loss.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

HPV genes in mice improve ear tissue healing by speeding up skin growth and repair.

One type of progenitor cell can maintain normal skin in mice.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

AP-2α and AP-2β proteins are essential for healthy adult skin and hair.

Notch-related genes play a key role in the development and cycling of hair follicles.

Plet-1 protein helps hair follicle cells move and stick to tissues.

PPARγ is essential for maintaining healthy skin, controlling inflammation, and ensuring proper skin barrier function.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

Blocking the mineralocorticoid receptor can help treat skin thinning caused by steroids.