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Meis1 is crucial for skin health and tumor development.

Indian Hedgehog helps control skin cell growth and protects against aggressive skin cancer.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

Certain skin cells near the base of hair muscles may help renew and stabilize skin, possibly affecting skin disorder understanding.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

Skin has specialized touch receptors that can tell different sensations apart.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Lgr5 is a marker for active, self-renewing stem cells in the intestine and skin, important for tissue maintenance.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Wnt signaling is crucial for skin wound healing and reducing scars.

The study found that a specific signaling pathway helps skin wounds heal faster but may lead to larger scars.

Different cell types work together to repair skin, and targeting them may improve healing and reduce scarring.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Blocking the mineralocorticoid receptor can help treat skin thinning caused by steroids.

One type of progenitor cell can maintain normal skin in mice.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

Skin stem cells maintain and repair the outer layer of skin, with some types being essential for healing wounds.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

Vitamin D helps protect skin, PSORS1 gene's risk interval for psoriasis is expanded, hair follicles can be generated from mouse cells, and interferon-γ may cause pigmented skin lesions.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Certain signals and genes play a key role in hair growth and regeneration, and understanding these could lead to new treatments for skin regeneration.

Fibroblasts, cells usually linked to tissue repair, also help regenerate various organs and their ability decreases with age. Turning adult fibroblasts back to a younger state could be a new treatment approach.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

Certain transcription factors are key in controlling skin stem cell behavior and could impact future treatments for skin repair and hair loss.