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Cancer treatments targeting specific cells and the immune system can cause skin, mouth, hair, and nail problems, affecting patients' quality of life and treatment adherence.

Activin activation in skin cells speeds up wound healing without affecting scar quality.

XEDAR triggers a specific signaling pathway in cells.

Mouse touch-sensitive nerve cells adjust their connections based on competition with other similar cells.

Cancer treatments targeting specific cells often cause skin, hair, and nail problems, affecting patients' lives and requiring careful management.

Chemotherapy causes complex changes in hair follicle cells that can lead to hair loss.

Urokinase, a type of protein, helps skin cells multiply faster, especially in newborn mice.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

Low-dose acitretin helps nail psoriasis, stem cells may treat scarring alopecia, Chinese men have lower baldness rates, lateral foldplasty is good for ingrown toenails, hair diameter helps diagnose female baldness, childhood trauma linked to alopecia areata, certain hair-weaving leads to scalp conditions in African American women, and new methods for hair research and understanding hair and sweat gland development were introduced.

Older mice healed wounds better but lost more weight and might have weaker immune systems afterward.

P-selectin is not the only factor that prevents scarring in fetal wound healing in mice.

A faulty KLHL24 gene leads to hair loss by damaging hair follicle stem cells.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Wounded skin cells can revert to stem cells and help heal.

Smart biomaterials that guide tissue repair are key for future medical treatments.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

MED1 is essential for normal hair growth and maintaining hair follicle stem cells.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Sirolimus and propranolol may reduce abnormal cell growth and improve lymphatic malformations in children.

Researchers successfully isolated and identified key markers of stem cell-enriched human hair follicle bulge cells.

TGF-β is crucial for controlling stem cell behavior and changes in its signaling can lead to diseases like cancer.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

The HR protein's role as a repressor is essential for controlling hair growth.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Hair greying is caused by oxidative stress damaging hair follicles and melanocytes.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

New understanding of the causes of primary cicatricial alopecia has led to better diagnosis and potential new treatments.