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The skin's basement membrane is specially designed to support different types of connections between skin layers and hair follicles.

Hair follicles can regrow in wounded adult mouse skin using a process like embryo development.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

The study introduced a new imaging technology to track skin healing and bone marrow cell activity over time.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

The gene Ascl4 is not necessary for the development of hair, teeth, or mammary glands.

Skin healing from blisters can delay hair growth as stem cells focus on repairing skin over developing hair.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

Stress increases nerve fibers and immune cell activity in mouse skin, possibly worsening skin conditions.

Newly divided skin cells quickly move to join skin structures due to tissue tension and specific signals.

The circadian clock affects skin stem cell behavior, impacting aging and cancer risk.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

In vivo confocal scanning laser microscopy is an effective, non-invasive way to study and measure new hair growth after skin injury in mice.

Skin lymphatic vessels are essential for hair growth.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Using polyethylenimine-DNA to deliver the hTERT gene can stimulate hair growth and may be useful in treating hair loss, but there could be potential cancer risks.

Hair follicle dermal stem cells are key for regenerating parts of the hair follicle and determining hair type.

Some stem cells in the body rarely divide, which could help create better treatments for diseases and aging.

Sebaceous glands can heal and regenerate after injury using their own stem cells and help from hair follicle cells.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

The document reports findings on genetic research, including ethical concerns about genome editing, improved diagnosis of mitochondrial mutations, solving inherited eye diseases, confirming gene roles in epilepsy, linking a gene to aneurysms, and identifying genes associated with age-related macular degeneration.

The mesenchyme can start hair growth, but the exact signal that causes this is still unknown.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Healthy mitochondria in skin cells are essential for proper hair growth and skin cell interaction in mice.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.