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Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

The Hairless gene is crucial for healthy skin and hair growth.

RXRα is crucial for proper immune response and links diet to immune function.

Deleting MED1 in skin cells causes hair loss and skin changes.

MED1 is essential for normal hair growth and maintaining hair follicle stem cells.

Vitamin D3 is important for bone health and may help treat various diseases beyond bone-related conditions.

Certain natural and synthetic compounds may help treat inflammatory skin diseases by targeting a specific signaling pathway.

Recent findings suggest that genetic factors, immune system issues, and skin cell defects might contribute to the development of hidradenitis suppurativa.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

Removing part of the vitamin D receptor stops vitamin D from working properly.

MicroRNAs are crucial for controlling skin development and healing by regulating genes.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

New treatments like nanotechnology show promise in improving skin cancer therapy.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Sebaceous glands can heal and regenerate after injury using their own stem cells and help from hair follicle cells.

Vitamin D receptor is crucial for bone health and mineral metabolism.

Basal cell carcinomas have much higher levels of Vitamin D3 receptors compared to healthy skin.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.

Certain herbal compounds might help prevent hair loss in menopausal women by activating Vitamin D receptors.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

HVDRR is caused by VDR gene mutations, leading to vitamin D resistance, treatable with high calcium doses, but alopecia remains permanent.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

Vitamin D Receptor is crucial for normal skin and hair growth.