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Lower SMAD2/3 activation predicts more severe skin cancer.

Blocking YAP/TAZ could be a new way to treat skin cancer.

EGFR helps control how hair grows and forms without needing p53 protein.

Polyamines are abundant in certain parts of rat hair follicles and may play a key role in hair growth.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

High LGR5 levels in glioblastoma indicate poor prognosis and are essential for cancer stem cell survival.

Runx1 affects hair growth, cancer development, and autoimmune diseases in epithelial tissues.

Apoptosis is crucial for healthy skin and treating skin diseases.

CXXC5 is a protein that controls cell growth and healing processes, and changes in its activity can lead to diseases like cancer and hair loss.

Overexpression of HDGF in melanocytes does not cause cancer.

CYLD mutations cause a variety of skin tumors with symptoms starting around age 16, and treatments are currently limited.

Sex-determining genes may affect male baldness.

Runx genes are important for stem cell regulation and their roles in aging and disease need more research.

Certain gene patterns in breast cancer are linked to how active hormone receptors are and could affect patient survival.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Patients with Bohring-Opitz syndrome and ASXL1 mutations need regular kidney ultrasounds to check for tumors.

Basaloid follicular hamartoma's appearance can be similar to other skin conditions, and diagnosis should consider both the look and behavior of the tumor.

Cancer can hijack the body's cell repair system to promote tumor growth, and targeting this process may improve cancer treatments.

The p53 protein has complex, sometimes contradictory functions, including tumor suppression and promoting cell survival.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Wnt and Shh signaling are key in noggin-induced tumors, and blocking them can slow tumor growth.

The research provides insights into hair follicle growth in forest musk deer by identifying key genes and pathways involved.

Understanding how EDC genes are regulated can help develop better drugs for skin diseases.

Four genes are potential markers for hair loss condition alopecia areata, linked to a specific type of cell death.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

Rat dermal papilla cells have unique genes crucial for hair growth.

Progesterone byproduct 5αP stimulates mammary tumor growth, but finasteride can suppress it.

Hair loss is significantly linked to lower levels of certain genes in hair follicles.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Researchers created a model to understand heart aging, highlighting the role of microRNAs and identifying key genes and pathways involved.