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Researchers found a specific T cell receptor linked to severe drug reactions like Stevens-Johnson syndrome when patients take carbamazepine.

New drug targets for hair loss treatment were identified by studying cell interactions in hair follicles.

Stem cell offspring help control their parent stem cells, affecting tissue health, healing, and cancer.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

People with HIV and low T cell counts have more hair and scalp problems.

Cell therapy shows promise for treating severe psoriasis but needs more research to confirm safety and effectiveness.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Baricitinib treatment helped reduce hair loss symptoms in mice by decreasing inflammation-related immune cells.

Assessing CD8+ T cell levels before surgery may improve vitiligo treatment outcomes.

Folliculotropic mycosis fungoides has unique molecular features and cell interactions that could guide targeted therapy.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

HIV patients with lower CD4 T cell counts often have more skin problems.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.

The COVID-19 pandemic led to fewer patient visits, more visits by older adults, an increase in cutaneous T-cell lymphoma cases, and a rise in noncosmetic procedures at a dermatology clinic.

Lichen planopilaris may be an autoimmune disease causing hair loss due to immune system issues in hair follicles.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Dihydrotestosterone (DHT) doesn't affect rat skin cell growth, but it does change cell cycle, protein levels, and other cell functions, potentially shortening hair growth cycle.

Simvastatin helps hair regrowth in autoimmune alopecia by directly affecting T cells.

MDSC-Exo can treat autoimmune alopecia areata and promote hair regrowth in mice.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Low serum complement levels in SLE patients don't always match with disease flares; monitoring C3 and C4 is useful, but cell-bound complement products might better indicate disease activity.

Severe alopecia areata involves higher levels of certain immune cells, which can be normalized with betamethasone.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Stem cell research and regenerative medicine have made significant advancements in treating various diseases and conditions.

IL-17 and certain immune cells are linked to more severe alopecia areata.