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COVID-19 can cause various skin lesions, which may result from the virus and immune response, and are not directly linked to illness severity.

Fisetin may help treat psoriasis and reduce skin inflammation.

Men are more likely to have severe respiratory viral infections like COVID-19 due to hormonal and genetic differences, while women generally have stronger immune responses.

A patient with Ivemark syndrome developed mixed type vitiligo after a hepatitis C infection, showing different treatment responses and immune cell involvement in the skin.

Inflammation linked to the immune response may play a role in causing various types of hair loss.

Nanoparticles can deliver vaccines through hair follicles, triggering immune responses and providing protection.

Lysophospholipids like LPA and S1P are important for hair growth, immune responses, and vascular development, and could be targeted for treating diseases.

Some patients with Alopecia Areata experience itch due to immune cells and enzymes that cause itching.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Alopecia areata involves immune system imbalances that may lead to depression and anxiety.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

Women with PCOS may have a higher risk of severe COVID-19 due to hormonal imbalances and vitamin D deficiency.

COVID-19 may cause temporary hair loss due to the body's immune response.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Protein tyrosine kinases are key in male pattern baldness, affecting skin structure, hair growth, and immune responses.

Different types of sun exposure damage skin cells and immune cells, with chronic exposure leading to more severe and lasting damage.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

Vitamin A affects hair loss and immune response in alopecia areata.

Astilbin can potentially calm overactive immune responses, like in Type 1 Diabetes, by suppressing certain cell activities and reducing inflammation.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

Stress increases nerve fibers and immune cell activity in mouse skin, possibly worsening skin conditions.

Alopecia areata severity and treatment response are linked to specific cytokine levels.

Unconventional lymphocytes are important for quick immune responses and healing of skin and mucosal barriers.

B2m-free HLA variants may be a new class of HLA important in immune responses and diseases.

New treatments for vitiligo may focus on protecting melanocyte stem cells from stress and targeting specific pathways involved in the condition.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Prolactin may be important for skin growth and immune function.

Using low-dose IL-2 to increase regulatory T cells might be a safe way to treat type 1 diabetes without severe side effects.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.