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Hair follicle development involves complex interactions between skin layers and cells, but many details are still unknown.

Skin cancer often starts from Lgr5+ progenitor cells.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

Skin organoids with NCSTN mutation show changes in hair follicle development and higher inflammation, key features of Hidradenitis Suppurativa.

The method quickly analyzes hair growth genes and shows that blocking Smo in skin cells stops hair growth.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

The Siah1 and Siah2 genes are active in mouse skin development and hair growth, especially right after birth.

Nestin marks cells that can become a specific type of skin cell in hair follicles of both developing and adult mice.

Excessive putrescine causes hair loss in transgenic mice by disrupting hair follicle development.

Researchers used a laser to create advanced skin models with hair-like structures.

CTCF protein is essential for skin and hair follicle development in mice.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.

The Hairless gene is crucial for healthy skin and hair growth.

Ectodysplasin signaling is crucial for skin appendage development, requiring specific doses and durations.

MOF controls key genes for skin development by regulating mitochondrial and ciliary functions.

mEphA1 receptor tyrosine kinase is important for skin and hair development and may play a role in certain diseases.

New skin substitutes for treating severe burns and chronic wounds are being developed, but a permanent solution for deep wounds is not yet available commercially.

Key skin cell regulators and gene organization changes are crucial for skin cell development and could help treat skin disorders.

Keratin genes change gradually during skin cell development and should be used carefully as biomarkers.

EZH2 is essential for hair growth and skin cell development.

Heterotypic cell contacts likely help hair matrix cells differentiate during mouse hair follicle development.

NGAL indicates abnormal skin cell differentiation.

Skin varies in thickness, color, and features due to complex genetic and cellular processes.

Artificial skin is improving wound healing and shows potential for treating different types of wounds.

Keratin gene expression helps understand different types of skin cells and their development, and should be used carefully as biological markers.

The symposium showed that stem cells are key for understanding and treating skin diseases and for developing new skin models and therapies.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.

Scientists created a lab-grown hair follicle model that behaves like real hair and could improve hair loss treatment research.

Different processes create patterns in skin and things like hair and feathers.

Neurotrophins are important for hair growth and could help treat hair loss.