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Excess male hormones in postmenopausal women can cause health issues and increase the risk of heart disease and diabetes; treatment depends on the cause.

Using two drugs together, Flutamide and 4-MA, is more effective for blocking male hormones than using each one alone.

The length of CAG repeats in the androgen receptor gene is linked to ovarian reserve but does not affect how the ovaries respond to stimulation.

Testosterone is crucial for development, growth, and various body functions in mammals.

Understanding how androgens work is key for creating new treatments for prostate issues and hair/skin conditions.

Combining flutamide and finasteride can reduce prostate weight and tumor growth, potentially benefiting treatments needing optimal DHT inhibition.

Ovarian Leydig cell tumors are hard to diagnose with just advanced imaging; expert ultrasound and clinical evaluation are essential.

No significant link between CAG repeat numbers and female pattern hair loss in Han Chinese population.

A genetic variant in the androgen receptor gene increases heart disease risk in women but not in men.

Doctors used a special blood sampling technique to diagnose a woman's rare ovarian tumor that was producing male hormones.

A new mutation linked to partial Androgen Insensitivity Syndrome and prostate cancer was found in a patient unhappy with their female gender assignment.

Androgen receptors in the mouse brain may explain cognitive and mood changes in prostate cancer treatment.

Testosterone therapy can help improve sexual function, mood, and bone density in women with low androgen levels, but more research is needed on long-term safety.

Using anabolic androgenic steroids can cause serious, lasting health problems in many parts of the body.

DHT is a strong androgen that may pose less risk to the prostate compared to testosterone.

Finasteride reduces testosterone conversion, progesterone lessens this, and levamisole enhances finasteride's effect.

The patient's high testosterone was reduced by a medication that suppresses gonadotropin.

Certain gene variations are linked to higher male hormone levels in Chinese women with PCOS and insulin resistance.

Androgen receptors are active in many tissues of both male and female mice, not just reproductive organs.

Anabolic steroids boost muscle growth, SARMs increase muscle mass and bone density without side effects, and myostatin inhibitors block a protein that stops muscle growth, but each has potential risks.

PCOS symptoms change with age; younger women have higher androgen levels and less metabolic issues, while older women face more metabolic challenges despite milder PCOS symptoms.

Testosterone replacement may improve sexual desire and bone health in women with low androgen levels, but more research is needed on its long-term safety.

Androgen self-administration might be controlled by membrane receptors, not nuclear ones.

The conclusion is that accurately identifying the cause of high androgen levels in women with PCOS is crucial and requires specific tests.

High androgen levels and genetic factors likely cause Becker's nevus and related symptoms.

Testosterone replacement may help post-menopausal women with androgen insufficiency, but more research is needed on its benefits and risks.

Menopausal women have higher androgen levels, which can cause symptoms like facial hair growth and low sexual desire.

The new progesterone derivatives effectively inhibit 5α-reductase and bind to the androgen receptor.

Obesity changes androgen levels in women with PCOS, leading to higher testosterone relative to androstenedione.

The same hormone can affect gene expression differently in various tissues, which could lead to new treatments for conditions like hair loss.