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Research 30 of 565
- Noggin is required for induction of the hair follicle growth phase in postnatal skin
- Shh maintains dermal papilla identity and hair morphogenesis via a Noggin–Shh regulatory loop
- Noggin overexpression inhibits eyelid opening by altering epidermal apoptosis and differentiation
- The Noggin null mouse phenotype is strain dependent and haploinsufficiency leads to skeletal defects
- Bone Morphogenetic Protein Antagonist Noggin Promotes Skin Tumorigenesis via Stimulation of the Wnt and Shh Signaling Pathways
- Expression characteristics of BMP2, BMPR-IA and Noggin in different stages of hair follicle in yak skin
- Effect of tofacitinib on the expression of noggin/BMP-4 and hair growth stimulation in mice
- The role of Wnt and Shh signaling systems in noggin-induced tumorigenesis
- Faculty Opinions recommendation of Bone morphogenetic protein antagonist noggin promotes skin tumorigenesis via stimulation of the Wnt and Shh signaling pathways.
- Role of the bone morphogenetic protein signalling in skin carcinogenesis. Effect of transgenic overexpression of BMP antognist Noggin on skin tumour development; molecular mechanisms underlying tumour suppressive role of the BMP signalling in skin.
- Impaired osteoblastic differentiation, reduced bone formation, and severe osteoporosis in noggin-overexpressing mice
- Inhibition of Bmp signaling affects growth and differentiation in the anagen hair follicle
- Bone Morphogenetic Protein Signaling Inhibits Hair Follicle Anagen Induction by Restricting Epithelial Stem/Progenitor Cell Activation and Expansion
- Molecular biology of hair morphogenesis: Development and cycling
- Transdifferentiation of corneal epithelium into epidermis occurs by means of a multistep process triggered by dermal developmental signals
- Laminin-511 is an epithelial message promoting dermal papilla development and function during early hair morphogenesis
- Bone morphogenetic protein signaling regulates the size of hair follicles and modulates the expression of cell cycle-associated genes
- Epidermal patterning and induction of different hair types during mouse embryonic development
- mTOR signaling promotes stem cell activation via counterbalancing BMP-mediated suppression during hair regeneration
- Bone morphogenetic protein (BMP) signaling controls hair pigmentation by means of cross-talk with the melanocortin receptor-1 pathway
- Bone Morphogenetic Protein Signaling Regulates Postnatal Hair Follicle Differentiation and Cycling
- Dynamic Signals for Hair Follicle Development and Regeneration
- Fibroblast growth factor 5-short (FGF5s) inhibits the activity of FGF5 in primary and secondary hair follicle dermal papilla cells of cashmere goats
- Conversion of the Nipple to Hair-Bearing Epithelia by Lowering Bone Morphogenetic Protein Pathway Activity at the Dermal-Epidermal Interface
- The balance of Bmp6 and Wnt10b regulates the telogen-anagen transition of hair follicles
- Transformation of amnion epithelium into skin and hair follicles
- The cellular basis of mechanosensory Merkel-cell innervation during development
- Induction of Skin-Derived Precursor Cells from Human Induced Pluripotent Stem Cells
- EDA and EDAR expression at different stages of hair follicle development in cashmere goats and effects on expression of related genes
- Impact of substrate stiffness on dermal papilla aggregates in microgels