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Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

Mice with the 'lanceolate hair' mutation have abnormal hair and skin similar to human Netherton's syndrome.

Hairless mice lack fur due to a genetic mutation affecting skin response, not hormone issues.

Platelet-rich plasma can help grow more mouse hair follicles, but it doesn't work for human hair follicles yet.

BMP2 needs periosteal tissue to help regenerate mouse middle finger bones within a specific time.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Extracts from Cercidiphyllum japonicum wood can stimulate mouse hair cell growth like common hair growth treatments.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

NF-κB is crucial for different stages and types of hair growth in mice.

Butin is effective in treating vitiligo in mice.

KLF4 is important for maintaining skin stem cells and helps heal wounds.

Altered retinoid metabolism in cicatricial alopecia suggests a balanced vitamin A diet may prevent the condition.

A mutation in mice causes hair loss and immune problems.

HPV genes in mice improve ear tissue healing by speeding up skin growth and repair.

A gene mutation in mice causes permanent hair loss and skin issues.

Non-thermal plasma treatment makes mouse skin thicker and increases growth factors without harming the tissue.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Hair loss in certain young mice is linked to a specific gene and can be caused by lack of iron.

Overexpressing SSAT in mice makes them highly sensitive to polyamine analogues, causing liver damage and high mortality.

Smad2/3-dependent TGF-β signaling increases during wound healing.

The research maps the complex development of early mouse skin, identifying diverse cell types and their roles in forming skin layers and structures.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

These methods help understand DNA changes in mouse skin.

The Itpr3 gene causes a specific hair pattern in mice.

Thiosulfate may help hair grow faster in mice and works well with a common hair growth treatment.

Mice with alopecia areata had wider lymphatic vessels in their skin.

The G(S) alpha subunit gene may help start hair follicle growth in newborn mice.

Scientists found markers called CD34 and CD200 that help identify stem cells in mouse and human hair follicles.

The research found changes in gene expression related to cell death in mouse skin that help understand hair follicle development and skin health.