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HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.

Finasteride exposure affects gene expression and anogenital distance in male rat fetuses.

Blocking certain immune signals can reduce skin damage from radiation therapy.

Key genes can rewire networks, changing skin appendage types.

The best long-lasting results in treating hair loss may be achieved through combination therapy, including treatments like finasteride, minoxidil, and platelet-rich plasma injections.

The document concludes that while some organisms can regenerate body parts, mammals generally cannot, and cancer progression is complex, involving mutations rather than a strict stem cell hierarchy.

Scientists are using clumps of special stem cells to improve organ repair.

The research identified key molecules that help hair matrix and dermal papilla cells communicate and influence hair growth in cashmere goats.

Botulinum toxin type A helps treat hair loss by stopping cell death in hair follicles through a process involving certain non-coding RNAs and a protein called Bax.

Lamivudine might reverse hair graying and needs more research for potential treatments.

Androgenic alopecia, a common hair loss condition, is linked to changes in androgen metabolism and genetics, and can be treated with finasteride and minoxidil, but these treatments are only fully effective in 10% of patients.

Different immune responses cause hair loss in scalp diseases, with unique patterns in scalp psoriasis possibly protecting against hair loss.

Chronic graft-versus-host disease in the skin shows strong Th1 immune response and unique barrier issues.

The study identified key genes that align with the cashmere growth cycle in goats, which could help improve cashmere production timing.

The research identified key genes that control the growth cycle of cashmere in goats, which could help improve cashmere goat breeding.

The studies identified key factors and potential treatments for skin and hair disorders.

TGFβ-2 may cause hair loss in androgenetic alopecia.

The document concludes that hair follicle regeneration involves various factors like stem cells, noncoding dsRNA, lymphatic vessels, growth factors, minoxidil, exosomes, and induced pluripotent stem cells.

Planarian regeneration begins with a specific gene activation caused by injury, essential for healing and tissue regrowth.

Minoxidil boosts hair growth by targeting adenosine and possibly sulfonylurea receptor 2B.

The testes control steroid production through complex interactions involving various cells and signaling molecules.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Hair follicles significantly enhance electroosmotic transport during iontophoresis.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Hormones and neuroendocrine factors control hair growth and color, and more research could lead to new hair treatment options.

The research explains how a human enzyme binds and processes its substrate, which could relate to its role in biological functions and hair loss.

Chronic scalp pain in trichodynia involves both body-wide and localized increased pain sensitivity.

Removing a methyl group from the ITGAV gene speeds up bone formation in a specific type of bone disease model.

Herbs might help with hair loss, but more research is needed to confirm their safety and effectiveness.

Sodium valproate helps skin healing by affecting GABA and histone deacetylase.