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Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

Type 1 Diabetes prevents hair growth by causing cell death in hair follicles.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

The nanofibers effectively treated infected diabetic wounds by killing bacteria and aiding wound healing without toxicity.

Recombinant keratin materials may better promote skin cell differentiation than natural keratin.

Hydrogel with M2-derived exosomes improves wound healing by slowly releasing exosomes that help reduce inflammation and promote tissue repair.

Acanthus ebracteatus Vahl. extract and verbascoside may help prevent hair loss and promote hair growth due to their anti-inflammatory properties and ability to protect against cell death.

The study found that sweat glands normally suppress immune responses, but this is disrupted in certain skin diseases, possibly contributing to their development.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Substance from human umbilical cord blood cells promotes hair growth.

Red light at 627 nm can safely trigger IL-4 release in skin cells, potentially helping treat inflammatory skin conditions.

The conclusion is that proper signaling is crucial for hair growth and development, and errors can lead to cancer or hair loss.

Blocking COX, especially COX-2, in the skin can reduce inflammation and pain and may help prevent skin cancer.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Human hair follicles can make and process prostaglandins, which may affect hair growth.

RANK is a key target in breast cancer treatment due to its role in tumor growth and bone metastasis.

Skin stem cells are maintained by signals from nearby cells and vary in their ability to renew and mature.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

Cholesterol-related compounds can stop hair growth and cause inflammation in a type of scarring hair loss.

Fat stem cells from diabetic mice can still help heal wounds.

Non-immune factors play a significant role in alopecia areata.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Placental mesenchymal stem cells and their conditioned medium significantly improve healing in local radiation injuries.

MOF controls skin development by regulating genes for mitochondria and cilia.

Implanted special stem cells from hair follicles helped heal wounds faster and with less scarring in mice.

Patients with Systemic Sclerosis have much higher levels of GDF-15, which could help predict organ involvement and guide treatment.