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The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Notch signalling helps skin cells differentiate and prevents tumors.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

Basal cell carcinomas may differentiate similarly to hair follicles and could be influenced by hair cycle-related treatments.

Hair follicle cells resist turning into skin cells.

A woman had a severe skin reaction from the drug imiquimod, used for skin cancer, highlighting the need for awareness of rare but serious side effects.

Radiation therapy successfully treated an elderly man's skin cancer and pre-cancerous scalp lesions.

Radiation therapy successfully treated an elderly man's skin cancer and pre-cancerous scalp lesions.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

Intu gene is crucial for hair follicle formation by helping keratinocytes differentiate through primary cilia.

Skin symptoms can indicate endocrine disorders and have various treatments.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

C/EBPalpha and C/EBPbeta are crucial for normal skin and oil gland cell development in adult mice.

Lipase H is important for hair follicle function and shaping hair fibers.

Vitamin D is important for skin health and can help treat psoriasis, atopic dermatitis, and vitiligo.

Hair follicles are essential for skin health, aiding in hair growth, wound healing, and immune function.

The risk of skin tumors becoming malignant depends on the specific skin cell type affected.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

2011 dermatology discussions highlighted stem cell hair treatments, new lichen planopilaris therapies, skin side effects from cancer drugs, emerging allergens, and the link between food allergies and skin issues.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

The cornified envelope is crucial for skin's barrier function and involves key proteins and genetic factors.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Non-coding RNAs are crucial for skin development and health.

The study concludes that regulating apoptosis could lead to new treatments for various skin and hair conditions.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

Inhibiting ODC can prevent UV-induced skin cancer.