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Understanding where cancer cells come from helps create better prevention and treatment methods.

Epigenetic mechanisms control skin development by regulating gene expression.

Activin activation in skin cells speeds up wound healing without affecting scar quality.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

Astrotactin2 affects hair follicle orientation and skin cell polarity.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Retinoids can prevent skin cancer in high-risk people but have side effects and require more research on dosing and effectiveness.

α-parvin is necessary for skin and hair growth and for the correct orientation of skin cells.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

The study found that sweat glands contain different types of stem cells that help with healing and maintaining healthy skin.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

The experiment showed that human skin grown in the lab started to form early hair structures when special cell clusters were added.

Researchers found that bulge cells from human hair can grow quickly in culture and have properties of hair follicle stem cells, which could be useful for skin treatments.

Bioactive molecules show promise for improving skin repair and regeneration by overcoming current challenges with further research.

Scientists successfully grew new hair follicles in regenerated mouse skin using mouse and human cells.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Sebaceous glands can heal and regenerate after injury using their own stem cells and help from hair follicle cells.

Targeting the HEDGEHOG-GLI1 pathway could help treat keloids.

Skin healing from blisters can delay hair growth as stem cells focus on repairing skin over developing hair.

The document concludes that understanding the sebaceous gland's development and function is key to addressing related skin diseases and aging effects.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

The research showed how melanocytes develop, move, and respond to UV light, and their stem cells' role in hair color and skin cancer risk.

Keeping β-catenin levels high in mammary cells disrupts their development and branching.

Experts met to improve care for ichthyosis patients in Spain.

TRPV3 could be a target for treating pain, skin disorders, and hair problems, but more research is needed to create effective drugs.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

The extracellular matrix affects where tumors can start in the body.