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A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

People with alopecia areata have more Th17 cells and fewer Treg cells, which may be key to the condition's development.

PPAR-γ is important for healthy hair and its problems, and more research on PPAR-γ treatments is needed.

A substance called 15-deoxy prostaglandin J2 can cause hair follicle cells to die, which might explain how prostaglandin D2 can lead to hair loss.

HAIR may cause fetal loss by triggering different cell death processes in the uterus and placenta.

Neural crest-derived progenitor cells in the cornea could help treat corneal issues without transplants.

Researchers created specific antibodies that detect a protein important in development and various conditions, and can be used for research and diagnosis.

Menthol boosts the hair growth effects of Phyllanthus niruri gel.

Targeting the HEDGEHOG-GLI1 pathway could help treat keloids.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

Softer hydrogels help wounds heal better with less scarring.

A new anti-baldness patch effectively treats hair loss by blocking enzymes linked to the condition.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

TNFα, IFNγ, and Substance P significantly affect prolactin levels in human skin, suggesting new treatments for skin and hair conditions.

Ptprq has multiple forms that change during inner ear development.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

Different markers are found in stem cells of the scalp's hair follicle bulge and the surrounding skin.

HSP90 and lamin A/C are crucial for hair growth and could be targets for treating hair loss.

Notch1 signaling is crucial for improving wound healing and skin regeneration by affecting stem cell behavior.

UVB exposure in human skin causes macrophages to produce more IL-10 and less IL-12, leading to immunosuppression.

Epidermal stem cells don't use gap junctions to communicate.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Using certain small proteins with a growth factor and specific materials can increase the creation of neurons from stem cells.

Dexamethasone increases the activity of androgen receptors in human skin cells, which may link it to certain types of hair loss.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

Using fat stem cells and blood cell-rich plasma together improves healing in diabetic wounds by affecting cell signaling.

Hair follicle dermal stem cells are key for regenerating parts of the hair follicle and determining hair type.

Stem cells from hair follicles in a special gel show strong potential for bone regeneration.