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Both HLA-B and MICA are independently linked to alopecia areata.

Hair proteins in preschool children and their mothers could indicate developmental changes and health status.

IL-9/IL-9R signaling can negatively affect human hair growth and may be a target for treating hair loss conditions.

The research developed methods to test drugs that could protect and restore hair follicle protection in a hair loss condition.

ULBP3 levels are higher in Tinea capitis patients and may help predict the disease's severity.

IL-15 helps protect hair follicles from immune attacks and encourages hair growth.

Scarred skin in lichen planopilaris loses immune cells due to a decrease in a specific protein in skin cells.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Some brain cancer cells avoid immune system detection, and certain treatments could target this to slow their growth; also, certain fat cell precursors help regenerate hair and skin after injury.

Free fatty acids in sebum boost skin's defense against acne by increasing antimicrobial peptides.

The new skin-targeted COVID-19 vaccine creates strong immune responses and could improve vaccination methods.

Coconut seed extract may effectively treat scabies in rabbits by killing mites and reducing inflammation.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

The technology can help diagnose and subtype autoimmune diseases by identifying specific autoantibodies.

Vitamin A affects hair loss and immune response in alopecia areata.

The protein interleukin-1 alpha helps regenerate hair follicles and increase stem cell growth in mice.

Androgenetic alopecia involves immune cell disruptions, especially increased CD4+ T cells around hair follicles.

Alopecia areata and vitiligo share immune system dysfunction but differ in specific immune responses and affected areas.

New treatments for ichthyosis, like protein replacement and gene therapy, show promise and may become standard care.

CO2 laser treatment for aging skin significantly changes immune system-related genes.

Targeting the protein Caveolin-1 might help treat a type of scarring hair loss called Frontal Fibrosing Alopecia.

KAP-depleted hair causes less immune response and is more biocompatible for implants.

The research suggests immune system changes and specific gene expression may contribute to male hair loss, proposing potential new treatments.

Immune changes and specific genes contribute to male hair loss.

Stress increases nerve fibers and immune cell activity in mouse skin, possibly worsening skin conditions.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

Lower CD200R1 on certain immune cells is linked to more severe rheumatoid arthritis and immune imbalance.

Psoriasis may be chronic because it lacks certain immune system controls that prevent overreaction.