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The NUDT15 gene variant causes severe side effects from azathioprine in some Indian patients.

The research provides a gene-based framework for hair biology, highlighting the Hippo pathway's importance and suggesting links between hair disorders, cancer pathways, and the immune system.

Found key genes affecting hair loss, immune response, and skin development; more research needed for better treatments.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

A patient with a new PLEC gene mutation showed symptoms of both muscular dystrophy and myasthenia gravis, which improved with steroid treatment.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

ADM scaffolds help skin heal by promoting a healing-type immune response.

A gene in Sebekia benihana, CYP-sb21, is needed for a specific reaction on the drug Cyclosporine A, which could be important for hair growth without affecting the immune system.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Advanced human skin models improve drug development and could replace animal testing.

The conclusion is that genetic changes in the glucocorticoid receptor can lead to conditions affecting stress response, immunity, and metabolism, requiring personalized treatment.

People with atopic dermatitis are more likely to develop other skin conditions due to shared genetics and immune pathways.

Genetic variants linked to ten skin diseases were found, showing both immune and non-immune factors play a role.

A boy's growth and immune problems were caused by a new mutation in the STAT5B gene.

Human prenatal skin develops an immune network early on that helps with skin formation and healing without scarring.

Scientists have found specific genes linked to different hair loss conditions, which could lead to new treatments.

DAVID syndrome is a condition with immune system and hormone deficiencies, needing early diagnosis to avoid serious complications.

Certain genetic variants linked to immune response increase the risk of alopecia areata in Taiwanese people.

TNC+ fibroblasts play a key role in skin inflammation by interacting with T cells.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Human hair follicle stem cells can be isolated using specific markers for potential therapeutic use.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

Skin organoids from stem cells could better mimic real skin but face challenges.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

Alopecia areata involves persistent gene abnormalities and immune activity, even in regrown hair, suggesting a risk of relapse.

Alopecia areata causes hair loss due to an immune attack on hair follicles, influenced by genetics and environment.