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Behçet's Disease may be caused by genetic and environmental factors leading to abnormal immune responses, and stress management and new treatments could improve patient outcomes.

Mesenchymal stem cells help improve wound healing by reducing inflammation and promoting skin cell growth and movement.

Special immune cells called Regulatory T cells help control skin inflammation and repair in various skin diseases.

Men are more likely to have severe respiratory viral infections like COVID-19 due to hormonal and genetic differences, while women generally have stronger immune responses.

Keratins are crucial for cell structure, growth, and disease risk.

Stress may trigger hair loss by affecting immune protection in hair follicles.

KAP-depleted hair causes less immune response and is more biocompatible for implants.

Keratins are important for skin cell health and their problems can cause diseases.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

IL-9 increases skin cell movement but decreases their ability to invade, and this effect is controlled by cell contractility, not by MMPs.

Low-Level Laser Therapy can stimulate healing and cell function, potentially leading to wider medical use.

Bead-jet printing of stem cells improves muscle and hair regeneration.

Wnt-signaling is regulated differently in skin cells and immune responses during wound healing.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Alopecia areata involves persistent gene abnormalities and immune activity, even in regrown hair, suggesting a risk of relapse.

Alopecia areata is caused by immune system issues, and JAK inhibitors might help treat it.

Chitosan nanofiber scaffolds improve skin healing and are promising for wound treatment.

Skin fat plays a key role in immune defense and healing beyond just storing energy.

Aging slows wound healing due to weaker cells and immune response.

The skin and thymus develop similarly to protect and support immunity.

Dermal macrophages might help regrow hair.

Psoriasis involves immune and genetic factors, and understanding these can improve treatments.

Fibroblasts, cells usually linked to tissue repair, also help regenerate various organs and their ability decreases with age. Turning adult fibroblasts back to a younger state could be a new treatment approach.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

Fat stem cells from diabetic mice can help heal skin wounds in other diabetic mice.

Using developmental signaling pathways could improve adult wound healing by mimicking scarless embryonic healing.

The research provides insights into hair follicle growth in forest musk deer by identifying key genes and pathways involved.

Beta-caryophyllene helps improve wound healing in mice, especially in females.

The shape of fibrous scaffolds can improve how stem cells help heal skin.