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Different Myc family proteins are located in various parts of the hair follicle and may affect stem cell behavior.

miR-31 regulates hair growth by controlling gene expression in hair follicles.

Gab1 protein is crucial for hair growth and stem cell renewal, and Mapk signaling helps maintain these processes.

Notch signaling disruptions can cause various skin diseases.

Researchers found genes linked to hair loss in male giant pandas.

Radiation therapy damages skin structure and immune function, causing inflammation and potential hair loss.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

A20 protein is crucial for normal skin and hair development.

Fixing DNA errors is crucial to prevent skin cancer.

Ribonucleotide excision repair is crucial to prevent skin cancer.

Ribonucleotide excision repair is crucial to prevent skin cancer.

Nrf2 helps protect skin from damage but too much can cause skin problems.

A gene mutation causes lanceolate hair in rats by disrupting hair shaft integrity.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Skin cell behavior is influenced by the tightness of nearby cells, affecting their growth and development.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

Increased HIF-1α is linked to the inflammation and severity of hidradenitis suppurativa, suggesting treatments that lower HIF-1α could help.

The mTOR pathway proteins are altered in the hair follicles of patients with Lichen Planopilaris and Frontal Fibrosing Alopecia.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

Keratin proteins are crucial for healthy skin, but mutations can cause skin disorders with no effective treatments yet.

Older skin has higher cancer risk due to inflammation and stem cell issues.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

α-parvin is necessary for skin and hair growth and for the correct orientation of skin cells.

The conclusion is that analyzing RNA from skin oils is a promising way to understand skin diseases.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.