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A new method helps isolate key hair components to study hair growth and loss.

VEGFR-2 is active in hair follicles, sebaceous glands, sweat glands, and skin on the human scalp.

Thioredoxin reductase 1 does not affect glucocorticoid receptor activity in hair follicle cells.

Intense pulsed light treatment mainly damages pigmented hair parts but spares stem cells, allowing hair to regrow.

The research identified specific genes that are active in the cells crucial for hair growth.

Exosomes from dermal papilla cells can help grow hair and might treat hair loss.

Morroniside may help hair grow and stay in its growth phase by affecting certain cell signals.

Certain genes are more active in baby scalp cells and can help grow hair when added to adult mouse skin cells.

Scalp hair follicle culture has limits for testing minoxidil's hair growth effects.

Ex vivo dermatoscopy may lower lab costs and improve diagnosis speed for hair loss biopsies.

Scalp biopsies from dermatomyositis patients show chronic hair loss without scarring, with mucin and blood vessel changes being very common.

Researchers developed a quick and easy way to get and grow cells from the base of human hair follicles.

RU58841 significantly increases hair growth rate and initiates more hair cycles, but doesn't affect hair thickness, suggesting it could be a new treatment for baldness.

Artemis protein may help control hair growth and health by influencing cell processes.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Balding hair follicle cells are smaller, grow less well, and need more effort to culture than non-balding cells.

Scalp areas that look normal in people with hair loss may still show signs of disease under a microscope.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

The study found that horizontal sections of scalp biopsies are better for analyzing hair loss, showing fewer hairs and more fine hairs in balding areas.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

Aging alone rarely causes significant hair loss; hormones are a bigger factor.

Lower growth factors linked to balding in androgenetic alopecia.

VEGFR-2 activation is likely involved in hair follicle growth, survival, and development.

The best long-lasting results in treating hair loss may be achieved through combination therapy, including treatments like finasteride, minoxidil, and platelet-rich plasma injections.

Human hair follicles can regenerate and recover after severe injury by going through a brief abnormal resting phase before growing again.

Cutting scalp biopsies in consecutive slices improves diagnosis of hair loss conditions.

A new nanoemulsion increases oxygen for hair cells, leading to better hair growth.

Human skin can make serotonin and melatonin.

Mutations in the LSS gene cause a rare type of hereditary hair loss.