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Certain immune system genes are linked to a higher risk of psoriasis and psoriatic arthritis, while others may offer protection.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

B2m-free HLA variants may be a new class of HLA important in immune responses and diseases.

Skin reactions to drugs are common and can be deadly, usually requiring stopping the drug and may be better prevented with genetic testing in the future.

Children's Serum Sickness-Like Reaction is often linked to antibiotics, especially amoxicillin, and involves symptoms like rash, fever, and joint issues.

Atopic dermatitis significantly lowers the quality of life for infants and their families.

Female hair loss can be diagnosed using specific trichoscopy criteria.

Some drugs can cause rare but potentially deadly skin reactions, and early treatment and avoiding the drug again are key.

The document concludes that reactivation of herpesviruses, especially HHV-6, is linked to severe symptoms and complications in drug-induced hypersensitivity syndrome.

Researchers found that most genes affecting drug responses are not fully covered by commercial SNP chips, suggesting the need for more comprehensive tools to optimize drug selection based on genetics.

A genetic marker linked to a type of hair loss was found in most patients studied.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

The document concludes that pregnancy and cancer share immune evasion tactics, but more research is needed before using checkpoint blockade immunotherapy in pregnant cancer patients to avoid harm to the placenta.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

Finasteride may cause lasting sexual and mental health issues, and genetic screening could help prevent them.

Certain HLA class II alleles increase or decrease the risk of alopecia areata.

Certain genetic markers, especially the MICA gene, are linked to alopecia areata.

Alopecia areata is an autoimmune condition causing hair loss due to the immune system attacking hair follicles, often influenced by genetics and stress.

Alopecia areata causes hair loss due to an immune attack on hair follicles, influenced by genetics and environment.

Certain genetic variants linked to immune response increase the risk of alopecia areata in Taiwanese people.

Alopecia areata has a complex genetic basis that was not fully understood as of 2001.

Some plant-based chemicals may help with hair growth, but more research is needed to confirm their effectiveness.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

Graft-versus-host disease is a complication where donor immune cells attack the recipient's body, often affecting the skin, liver, and gastrointestinal tract.

Somatostatin may help protect hair follicles from immune attacks.

Some systemic treatments work for nail psoriasis but can have serious side effects.

More research is needed to understand the genetic causes of Alopecia areata to develop better treatments.

Hair loss from Alopecia Areata is caused by both genes and environment, with several treatments available but challenges in cost and relapse remain.

Targeting the protein Caveolin-1 might help treat a type of scarring hair loss called Frontal Fibrosing Alopecia.

Some drugs can cause skin, nail, and hair problems, which are important for healthcare professionals to recognize and report.